Boletín Electrónico CDIC. No. 4 marzo 2008
Este boletin se distribuye a 339 usuarios de CDIC.

Ley obliga el depósito de artículos de investigaciones financiadas por el NIH


Ley firmada en diciembre de 2007 por el Presidente Bush hace obligatoria la publicación en el repositorio PubMed Central de toda investigación financiada por el NIH y publicada en revistas con procedimientos de evaluación por pares. La legislación impulsa el movimiento de Acceso Abierto y el acceso a la información científica en los países menos favorecidos. Dos modelos de Acceso Abierto prevalecen: 1) el de la creación de revistas científicas o transformación de las ya existentes en Acceso Abierto y 2) el de depósito de los artículos después de su aprobación en repositorios de acceso abierto. Actualmente hay tres mil revistas de acceso abierto indizadas en el DOAJ. La red SciELO, lanzada hace 10 años bajo el liderazgo de FAPESP y BIREME/OPS/OMS, publicó en diciembre de 2007 cerca de 500 revistas (11% total de títulos del DOAJ). Ya el OpenDOAR registra en diciembre de 2007 más de mil repositorios institucionales. En ciencias de la salud se destacan dos repositorios PubMed Central, uno del NIH, Estados Unidos y otro del Welcome Trust, Reino Unido

BIREME, Sao Paulo, Brasil.

Newsletter BVS 077 09/ENERO/2008

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“En el ámbito científico los autores no reciben una remuneración monetaria directa, 
  no participan en los beneficios económicos que la comercialización de la revista pueda generar”

 “Es difícil justificar que el conocimiento, pagado, producido y validado con recursos públicos,
   pueda acabar siendo propiedad de unas empresas”

[Fuente]:   El profesional de la información, 14(5), 2005.


LILACS alcanza resultados históricos


Fuente de información de registro e indización de la literatura científica y técnica, LILACS (Literatura Latinoamericana y del Caribe en Ciencias de la Salud) alcanza un nuevo nivel de contribución en el ingreso de referencias bibliográficas de documentos científicos y técnicos como artículos, monografías, trabajos de congresos, tesis y documentos gubernamentales. El trabajo cooperativo en red con un creciente compromiso de los centros cooperadores contribuyó para este avance histórico, además de las facilidades que aportan las innovaciones en la metodología y tecnología utilizadas para alimentar y publicar LILACS.

BIREME, Sao Paulo, Brasil

Newsletter BVS 078 29/febrero/2008

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Overweight, obesity, and mortality from cancer in a prospectively studied cohort of U.S. adults.


Calle EE, Rodriguez C, Walker-Thurmond K, Thun MJ.

Department of Epidemiology and Surveillance Research, American Cancer Society, Atlanta 30329, USA. jcalle@cancer.org

BACKGROUND: The influence of excess body weight on the risk of death from cancer has not been fully characterized. METHODS: In a prospectively studied population of more than 900,000 U.S. adults (404,576 men and 495,477 women) who were free of cancer at enrollment in 1982, there were 57,145 deaths from cancer during 16 years of follow-up. We examined the relation in men and women between the body-mass index in 1982 and the risk of death from all cancers and from cancers at individual sites, while controlling for other risk factors in multivariate proportional-hazards models. We calculated the proportion of all deaths from cancer that was attributable to overweight and obesity in the U.S. population on the basis of risk estimates from the current study and national estimates of the prevalence of overweight and obesity in the U.S. adult population. RESULTS: The heaviest members of this cohort (those with a body-mass index [the weight in kilograms divided by the square of the height in meters] of at least 40) had death rates from all cancers combined that were 52 percent higher (for men) and 62 percent higher (for women) than the rates in men and women of normal weight. For men, the relative risk of death was 1.52 (95 percent confidence interval, 1.13 to 2.05); for women, the relative risk was 1.62 (95 percent confidence interval, 1.40 to 1.87). In both men and women, body-mass index was also significantly associated with higher rates of death due to cancer of the esophagus, colon and rectum, liver, gallbladder, pancreas, and kidney; the same was true for death due to non-Hodgkin's lymphoma and multiple myeloma. Significant trends of increasing risk with higher body-mass-index values were observed for death from cancers of the stomach and prostate in men and for death from cancers of the breast, uterus, cervix, and ovary in women. On the basis of associations observed in this study, we estimate that current patterns of overweight and obesity in the United States could account for 14 percent of all deaths from cancer in men and 20 percent of those in women. CONCLUSIONS: Increased body weight was associated with increased death rates for all cancers combined and for cancers at multiple specific sites.

NEJM, 2003 Apr 24;348(17):1625-38.

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International Research Conference on Food, Nutrition & Cancer


Physical Activity and Cancer Prevention: Etiologic Evidence and Biological Mechanisms1 ,2

Christine M. Friedenreich3 and Marla R. Orenstein

Division of Epidemiology, Prevention and Screening, Alberta Cancer Board, Calgary, Alberta, Canada, T2N 1N3
To whom correspondence should be addressed. E-mail: chrisf@cancerboard.ab.ca.

Scientific evidence is accumulating on physical activity as a means for the primary prevention of cancer. Nearly 170 observational epidemiologic studies of physical activity and cancer risk at a number of specific cancer sites have been conducted. The evidence for decreased risk with increased physical activity is classified as convincing for breast and colon cancers, probable for prostate cancer, possible for lung and endometrial cancers and insufficient for cancers at all other sites. Despite the large number of studies conducted on physical activity and cancer, most have been hampered by incomplete assessment of physical activity and a lack of full examination of effect modification and confounding. Several plausible hypothesized biological mechanisms exist for the association between physical activity and cancer, including changes in endogenous sexual and metabolic hormone levels and growth factors, decreased obesity and central adiposity and possibly changes in immune function. Weight control may play a particularly important role because links between excess weight and increased cancer risk have been established for several sites, and central adiposity has been particularly implicated in promoting metabolic conditions amenable to carcinogenesis. Based on existing evidence, some public health organizations have issued physical activity guidelines for cancer prevention, generally recommending at least 30 min of moderate-to-vigorous intensity physical activity on =" src="/emath/ge.gif" border=05 d/wk. Although most research has focused on the efficacy of physical activity in cancer prevention, evidence is increasing that exercise also influences other aspects of the cancer experience, including cancer detection, coping, rehabilitation and survival after diagnosis.

J. Nutr.  3456S-3464S, 2002

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